Cystic Fibrosis Reversed – Amazon Kindle and CreateSpace book. Treat CF with breathing retraining and more oxygen in body cells.
Cystic fibrosis life expectancy has been growing for several decades since 1930s, when most babies with CF died before their first birthday. Later, in the 1950s, cystic fibrosis life expectancy was less than 10 years. Soon after, due to use of various therapies, it increased from about 14 years (in the 1980s) up to about current 35-37 years. Some researchers predict that cystic fibrosis life expectancy for contemporary babies can be up to 50 or more years.
Cystic fibrosis major symptoms can be eliminated by any person who achieves normal breathing parameters. Therefore cystic fibrosis treatment should target one main goal: to increase body O2 content. This is exactly the key problem in adults and children with cystic fibrosis that limits their cystic fibrosis life expectancy.
O2 in cells is the crucial factor in cystic fibrosis since the tiny pumps that transport sodium and chloride ions to create mucus in the respiratory tract and digestive system require oxygen to do the job correctly. Low O2 level in cells makes these pumps malfunction. And this is effect is established even in ordinary people without the CFTR gene. People with CF (cystic fibrosis) experience the same effect (poor work of tiny pumps causing more viscous mucus), but this effect is amplified by their CFTR gene making mucus, in conditions of tissue hypoxia, even thicker and dryer.
That causes opportunistic infections in the GI tract, airways, and lungs. In addition, low O2 or tissue hypoxia causes oxidative stress and generations of free radicals due to anaerobic respiration in cells. This suppresses the immune system and worsens infections. They become chronic reducing cystic fibrosis life expectancy.
Adults and children with cystic fibrosis develop chronic GI problems due to advance of Candida Albicans, Helicobacter Pylori, and other bacteria, fungi and pathogens. Similar effects take place in the respiratory problems resulting in frequent respiratory infections with other symptom that are different and individual and can include chronic cough, inflammation of airways, extra mucus, development of allergies and wheezing, poor fitness, problems with sleep and many other effects.
The solution to all these problems is to increase body O2 content that directly improves cystic fibrosis life expectancy. Why adults and children with CF have reduced O2 content in cells? This is due to their too heavy and fast breathing at rest. Numerous studies showed that people with cystic fibrosis breathe about twice heavier for their minute ventilation at rest.
Overbreathing is also called hyperventilation. It destroys tissues of the lungs due to lack of CO2 that has protective effects on lung tissues and performs key roles in O2 transport. Indeed, people with overbreathing either destroy their lungs (as it happens in severe asthma, COPD and emphysema) or suffer from low arterial CO2 when lungs are not involved yet.
Low arterial CO2 causes devastating effects on O2 delivery to cells. First of all, CO2 expands arteries since it is the most potent known vasodilator. Therefore, low arterial CO2 immediately leads to reduced perfusion (or circulation) to all vital organs, including the brain, heart, lungs, stomach, colon, and so forth. In addition, low CO2 leads to the suppressed Verigo-Bohr effect. This means that less oxygen is released in tissues by red blood cells due to lack of CO2. This effect worsen hypoxia in people with cystic fibrosis (http://www.normalbreathing.com/cystic-fibrosis1.php) causing more problems with the CFTR gene and normal mucus formation.
You can find out all studies and details of successful treatment of cystic fibrosis from the Amazon Kindle book “Cystic Fibrosis Life Expectancy: 30, 50, 70…” by Dr. Artour Rakhimov (NormalBreathing.com). http://www.amazon.com/Cystic-Fibrosis-Life-Expectancy-ebook/dp/B00793UMNQ/ – This book has a PDF version too.
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